Periodontal Diseases: Gingivitis and Periodontitis — Diagnosis and Management
Dental Health · Oral and Dental Conditions Periodontal Diseases: Gingivitis and Periodontitis — Diagnosis and Management Periodontal diseases affect the tissues that surround and support the teeth. They include gingival inflammation, most commonly plaque-induced gingivitis, and periodontitis, in which inflammation is associated with loss of the tooth’s supporting attachment and alveolar bone. Gingivitis is often reversible when the cause is controlled. Periodontitis can usually be stabilised and managed, but lost attachment and bone are not routinely restored by simple cleaning alone. Early recognition, accurate examination, effective daily plaque control, risk-factor management and professional follow-up help protect oral function and reduce the risk of tooth loss. This study guide expands the course slide resources and applies current periodontal terminology. It is written for health-science learners and does not replace supervised clinical training, a dental diagnosis or local Ugandan protocols. A trained dental professional should assess individual patients and decide treatment according to the findings, available facilities, patient preferences and referral pathways. Learning objectives By the end of this lesson, you should be able to: define the periodontium and common periodontal terms; distinguish gingivitis from periodontitis; describe causes, risk factors, clinical features and complications; take a focused periodontal history; outline screening, periodontal charting and indicated radiography; explain the stepwise principles of management; counsel patients on prevention and risk reduction; and recognise situations that need urgent dental or specialist referral. 1. The periodontium and key terms The periodontium is the group of tissues that support and attach a tooth. Its main parts are the gingiva, periodontal ligament, cementum covering the tooth root and alveolar bone. Disease may affect the gingiva alone or damage deeper supporting structures. Gingiva: the gum tissue surrounding the teeth and covering the alveolar processes. Healthy gingiva is generally firm and closely adapted to the tooth, though colour and appearance vary with pigmentation and individual anatomy. Dental plaque biofilm: a structured community of microorganisms in a matrix that forms on teeth and restorations. Persistent biofilm at the gingival margin can provoke inflammation in a susceptible host. Calculus (tartar): mineralised plaque. It is not itself the primary cause of periodontitis, but its rough surface can retain biofilm and it cannot be removed by ordinary brushing once firmly deposited. Gingivitis: inflammation confined to the gingiva, typically causing redness, swelling and bleeding on gentle probing or brushing, without periodontitis-related clinical attachment loss or bone loss. Periodontal pocket: an abnormally deepened space between a tooth and the gingiva. Pocket depth must be interpreted with gum position, clinical attachment level, bleeding and other findings; a deep reading alone does not establish the full diagnosis. Probing depth (PD): the distance from the gingival margin to the base of the sulcus or pocket, measured with a periodontal probe. Clinical attachment level (CAL): the distance from a fixed tooth landmark, usually the cementoenamel junction, to the base of the pocket. CAL helps identify loss of tooth-supporting attachment. Gingival recession: movement of the gum margin away from the crown, exposing part of the root. Recession can result from several causes and is not, by itself, proof of periodontitis. Bleeding on probing (BOP): bleeding observed after gentle probing. It is a sign of inflammation at a site and should be recorded alongside plaque and pocket measurements. Furcation involvement: loss of supporting tissues between the roots of a multi-rooted tooth. Tooth mobility: movement of a tooth beyond normal physiological movement. It can have periodontal and non-periodontal causes and is assessed in context. Suppuration: discharge of pus from the gingival sulcus or pocket, suggesting active infection or an abscess and requiring professional assessment. 2. Classification and patterns of disease Current international classification distinguishes periodontal health, gingival diseases and conditions, periodontitis, and other conditions affecting the periodontal tissues. Older teaching slides may use labels such as “chronic” and “aggressive” periodontitis. In the 2017 World Workshop classification, these former categories are grouped within periodontitis and described using stage and grade, while recognising necrotising disease and periodontitis as a manifestation of certain systemic diseases as distinct forms. Condition Main feature Why it matters Plaque-induced gingivitis Gingival redness, swelling and bleeding related to dental biofilm; no periodontitis-related attachment or bone loss. Usually reversible when plaque is effectively removed and contributing factors are addressed. Non-plaque-induced gingival disease Gingival lesions or inflammation related to infection, immune or mucocutaneous disease, allergy, trauma or another cause. May persist despite plaque removal; identify the cause and refer when findings are atypical or persistent. Periodontitis Inflammatory destruction of tooth-supporting attachment and alveolar bone, often with pockets, recession, bleeding or mobility. Can lead to progressive support loss and tooth loss; requires professional treatment and ongoing supportive care. Necrotising periodontal disease Acute painful gingival necrosis, often with punched-out interdental papillae, spontaneous bleeding and halitosis; systemic symptoms may occur. Needs prompt assessment, pain control, careful local treatment and evaluation of predisposing factors or systemic illness. Periodontal abscess Localised accumulation of pus in the gingival wall of a periodontal pocket, often with local pain, swelling and tenderness. May cause rapid tissue destruction and can spread; urgent dental assessment is required. Periodontitis associated with a systemic condition Periodontal destruction occurring in the setting of a systemic disorder that affects the periodontal apparatus. Document relevant medical conditions and coordinate care with the appropriate clinician. 3. Causes and pathogenesis Dental plaque biofilm at and below the gingival margin triggers a host inflammatory response. When plaque remains undisturbed, the microbial community can become more disease-associated. In gingivitis, inflammation is limited to the gum tissue. In susceptible people, persistent inflammation may involve destruction of periodontal ligament fibres and resorption of alveolar bone. The depth and distribution of damage vary across teeth and patients; the amount of visible plaque alone does not predict severity. Calculus, overhanging or poorly contoured restorations, crowded teeth, appliances and other plaque-retentive features can make cleaning difficult. Systemic and behavioural factors modify inflammation and progression. Smoking can mask bleeding while worsening periodontal risk and treatment response. Diabetes, especially when glycaemic control is poor, is an important modifying factor; periodontal inflammation and diabetes can influence each other, but

